Continues from part 6

My Personal Problems: Part 7

All rights Reserved

Egret Publishing Inc. August 2002

 

 

 

Section One: Various PCR Methods

Most Paucibacillary Tuberculoid Cases Tested Negative by means of PCR

 

Many PCR methods have been developed and published, with different performances. The one used by the Provincial Laboratory in my case, developed by D. Williams and associates, gives positive signal at 100 bacteria but not at 10. wil  There are others, claimed by its authors to be more sensitive and just as specific. According to Polymerase Chain Reaction for the Detection of Mycobacterium leprae by Rudy A. Hartskeerl et al, M. leprae could be detected specifically with a detection limit approximating one bacterium. har  Most methods have detection limits at 10 bacteria and up. The one used by the Provincial Laboratory is the least sensitive of the lot. The report from the Provincial Laboratory stated that it was for research purposes only. Most paucibacillary such as tuberculoid patients diagnosed of Hansen’s disease did not test positive for Hansen’s bacterium by means of PCR (with detection limit of 10 bacteria). Some were diagnosed clinically and some were diagnosed histopathologically. job

 

Section Two: Proper Perspective about PCR Methods

 

PCR methods are several hundred times more sensitive than the conventional microscopic method but it is still not sensitive enough to accomplish the task, detecting Hansen’s bacterium present in small number. I was misled by the overblown optimism of some authors, until I found some papers, describing PCR methods (see Section One) in practice, regarding detection of Hansen’s bacterium in paucibacillary patients. Before the advent of PCR methods, it would be almost impossible to observe Hansen’s bacterium in the case of tuberculoid Hansen’s disease (by the conventional staining-microscopic technique). The link between tuberculoid and Hansen’s bacterium might have been accomplished by analogy. Tuberculoid type was similar to other types of Hansen’ disease, which were linked to Hansen’s bacterium. Therefore tuberculoid must be linked to Hansen’s bacterium as well. Results of antibiotics treatments of tuberculoid patients may have confirmed the assumption.

 

Most cases of tuberculoid Hansen’s disease are diagnosed by means of clinical and histopathological methods. Clinical and histopathologic methods are more sensitive than PCR methods as far as tuberculoid or paucibacillary cases are concerned.

 

Section Three: If not PCR, What Methods Can Solve the Problem?

 

At this stage of development, PCR methods may not be able to detect Hansen’s bacterium in ALS patients if it is present. Mass-spectrometric methods are yet to be developed. The best bet is to emulate diagnosis of paucibacillary cases such as tuberculoid Hansen’s disease.

 

In similar way, this can be done in the cases of ALS and tuberculoid Hansen’s disease, considering the similarity of their symptoms, such as foot drop, cramp, spasm and muscle weakness. Most ALS patients have difficulty in swallowing. Lepromatous type of Hansen’s disease can cause throat problems such as difficulty in swallowing (source: conversation with the infectious disease specialist). The observation of the antigens of Hansen’s bacterium in the central nervous system of Hansen’s disease patients (see Notes and Comments #2) indicates the central nervous system can be afflicted by the pathogen. If antibiotics stop ALS, it will be an evidence to support the argument that ALS is caused by infection. As analytical techniques, such as PCR and mass-spectrometric methods, are improved, Hansen’s bacterium will be detected in ALS patients. 

 

Minocycline and tetracycline are used to treat acne but have they identified the pathogen causing the problem? These drugs work on acne in practice and also work in cases of Hansen’s disease. Acne is not a life-threatening condition. I am suffering from something (ALS?), which may lead to serious or life-threatening situations.

 

Section Four: an Interesting Conversation

 

Recently I visited an eye doctor. In the waiting room, a few elderly ladies were engaged in animated conversation, about the deaths and of what diseases of their friends and relatives. One lady said, “After so much expenses and time for research, all those diseases are still incurable.” It looks like that ordinary people are beginning to wonder. If one is looking for oranges in a lemon grove, he will not find it. Researchers may be searching in the wrong place. Big increases in the numbers of cases of these diseases indicate infection as a probable cause.

 

In view of increasing number of people suffering from incurable diseases, there should be no stones left unturned. All probable causes of the diseases should be investigated. The attitude of the medical authorities seems very strange, regarding infection as a cause of these diseases. The lack of curiosity is amazing.

 

Ching-Chee Chan, PhD

 

August 2, 2002

 

 

 

References

 

wil. Williams DL, Gillis TP, Booth RJ, Looker D and Watson JD. The use of a specific DNA and polymerase chain reaction for the detection of Mycobacterium leprae. JID 1990; 162 (July): 193-200.

 

har. Hartskeerl RA, De Wit MYL and Klatser PR. Polymerase chain reaction for the detection of Mycobacterium leprae. Journal of General Microbiology 1989; 135 (September): 2357-2364.

 

job. Job CK, Jayakumar J, Williams DL and Gillis TP. Role of Polymerase Chain Reaction in the Diagnosis of Early Leprosy. Int J Lepr 1997; 65(4): 461-464.

 

 

 

 

Readers are welcome to e-mail me to discuss relevant problems.

 

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